Hello parents and family members of autistic children:
I have found some interesting information about the RNA viruses associated with autism. RNA viruses for HIV and cancer are inhibited by Tetrasilver Tetroxide US Pat 5676977 and US Pat 6485755 respectively.
Association of Autism with Polyoma Virus Infection in Postmortem Brains.
Journal Neurovirology, 2010 March – Lintas C, Altieri L, Lombardi F, Sacco R, Persico AM.Laboratory of Molecular Psychiatry and Neurogenetics, University Campus Bio-Medico, Rome, Italy.[1]
These viruses were found in the brain DNA of 15 autistic children in Italy who died by 2010 but had diagnoses of autism. How did the JC virus from the US and the BK virus from the US survive from 1971 until 2010 for 39 years? Tissue decomposes when someone dies usually within 3 months in a moist climate {see http://fac.utk.edu/}
Interestingly to note there were a total of FIVE herpes viruses and two people viruses from patients who died in 1971 in the US! Lastly Lintas found the AIDS or HIV virus from green monkeys’ kidneys. This virus has been called the SV-40 virus and first was named the SE Polyoma virus for Dr. Bernice Eddy, PhD, and Dr Sarah Stewart, MD, PhD who found it in polio vaccine complement used in the 1950s through 1963. They found that the SE Polyoma virus could cause cancer in many animal species and feared that it would cause cancer in humans.[2]
Carla Lintas found these RNA viruses: cytomegalovirus (CMV), Epstein-Barr virus (EBV), herpes simplex virus type 1 (HSV1), herpes simplex virus type 2 (HSV2), human herpes virus 6 (HHV6), BK virus (BKV), JC virus (JCV), and simian virus 40 (SV40) in genomic DNA
- The most prevalently occurring viruses were RNA Herpes Viruses to include Herpes 1, 2, 4 EBV, 5 CMV, and 5 measles roseola.
- Herpes I is herpes simplex virus type 1
- HSV2 herpes simplex virus type 2 (HSV2)
- Epstein-Barr virus (EBV) herpes virus 4 (HV4)
- cytomegalovirus (CMV) human virus 5 (HV5)
- human herpes virus 6 (HHV6) also known as roseola or Measles
- BK virus (BKV)
- JC virus (JCV)
- Green Monkey Kidney virus simian virus 40 (SV40) found by Maurice Hilleman, PhD of Merck fame in the late 1950s following research by Drs. Eddy and Stewart
- bkvirus – Nephropathy from BK virus (BKV) infection is an evolving challenge in kidney transplant recipients. (cjasn.asnjournals.org/content/2/Supplement_1/S36.full)
Who is BK of the bkvirus and what family is it in?
The BK virus was first isolated in 1971 from the urine of a renal transplant patient, initials B.K.[3]The BK virus is similar to another virus called the JCV since their genome sequences share 75% homology. [Essentially these viruses are very identical they share 75% of the same genome sequences but BK and JC were not related humans.] Polyomaviridae is the bkvirus family. ] Polyoma is the genus of the bkvirus and jcvirus. Both of these viruses can be identified and differentiated from each other by carrying out serological tests using specific antibodies or by using a PCR based genotyping approach.
Who is JC of the jcvirus?
The jcvirus was from a patient with progressive multifocal leukoencephalopathy (PML).[4]
Why are the animal and herpes RNA viruses mentioned in the work of Carla Lintas so significant?
Where else are so many and varied animal viruses present to make cells infected with Multiple Sclerosis? In the cell of a woman patient with multiple sclerosis cultured in US PAT 6582703 See Figure 45. A total of 18 different animal viruses were found in the Multiple Sclerosis cell of an infected woman that were tissue cultured for sale by Perron and Jean Marie Seigneurin co-holders of US Patent 6582703 In FIGURE 45 RNA Viruses from Asian Indian Gibbon Ape, West African Green Monkey, South African Baboon, Chimp, Central/South American Squirrel Monkey, Icelandic Sheep Nazi Maedi VISNA virus, Cow Lymphoma, Horse Leukemia, + Simian Foamy Virus etc. are mentioned.
18 Animal Viruses in Multiple Sclerosis tissue culture infection
http://www.patentstorm.us/patents/6582703/fulltext.html
What does a tetrasilver tetroxide recipient look like after being cured of HIV and having no RNA viruses detectable in the blood?
http://www.facebook.com/media/set/?set=a.247457775273179.65761.100000268761354&type=3
Autism is a highly heritable behavioral disorder. Yet, two decades of genetic investigation have unveiled extremely few cases that can be solely explained on the basis of de novo mutations or cytogenetic abnormalities. Vertical viral transmission represents a nongenetic mechanism of disease compatible with high parent-to-offspring transmission and with low rates of disease-specific genetic abnormalities. Vertically transmitted viruses should be found more frequently in the affected tissues of autistic individuals compared to controls. Our initial step was thus to assess by nested polymerase chain reaction (PCR) and DNA sequence analysis the presence of cytomegalovirus (CMV)[Herpes Virus V], Epstein-Barr virus (EBV)[Herpes Virus iV], herpes simplex virus type 1 (HSV1), herpes simplex virus type 2 (HSV2), human herpes virus 6 (HHV6)[Herpes Virus known as roseola or measles such as measles vaccine], BK virus (BKV)[from BK’s kidney], JC virus (JCV)[from JC’s brain leukoencechalopathy], and simian virus 40 (SV40)[from a West African green monkey kidney] in genomic DNA extracted from postmortem temporocortical tissue (Brodmann areas 41/42) belonging to 15 autistic patients and 13 controls. BKV, JCV, and SV40 combined are significantly more frequent among autistic patients compared to controls (67% versus 23%, respectively; P < .05). The majority of positives yielded archetypal sequences, whereas six patients and two controls unveiled single-base pair changes in two or more sequenced clones. No association is present with the remaining viruses, which are found in relatively few individuals (N </= 3). Also polyviral infections tend to occur more frequently in the brains of autistic patients compared to controls (40% versus 7.7%, respectively; P = .08). Follow-up studies exploring vertical viral transmission as a possible pathogenetic mechanism in autistic disorder should focus on, but not be limited to, the role of polyoma viruses. PMID: 20345322″
1 Association of Autism with Polyoma Virus Infection in Postmortem Brains. Journal Neurovirology, 2010 March – Lintas C, Altieri L, Lombardi F, Sacco R, Persico AM.Laboratory of Molecular Psychiatry and Neurogenetics, University Campus Bio-Medico, Rome, Italy. http://umanitoba.ca/faculties/medicine/medical_microbiology/SeminarPDF/polyomavirus_infection_Stachowiak_Nov2010.pdf or http://www.ncbi.nlm.nih.gov/pubmed/20345322
2 EDDY BE, STEWART SE. Characteristics of the SE polyoma virus. Am J Public Health Nations Health. 1959 Nov;49:1486–1492. http://www.ncbi.nlm.nih.gov/pubmed/13819251
3 Gardner SD, Field AM, Coleman DV, Hulme B (June 1971). “New human papovavirus (B.K.) isolated from urine after renal transplantation”. Lancet 1 (7712): 1253–7. PMID 4104714.
4 BL, Walker DL et al (1971). “Cultivation of papova-like virus from human brain with progressive multifocal leucoencephalopathy”.Lancet 1 (7712): 1257–60. doi:10.1016/S0140-6736(71)91777-6. PMID 4104715.
Cynthia Barrington Tetrasilver 